Prior facial motor neuron injury elicits endogenous T cell memory: Relation to neuroregeneration
نویسندگان
چکیده
منابع مشابه
Repair of Spinal Cord Injury by Co-Transplantation of embryonic Stem Cell-Derived Motor Neuron and Olfactory Ensheathing Cell
Background: The failure of regeneration after spinal cord injury (SCI) has been attributed to axonal demyelination and neuronal death. Cellular replacement and white matter regeneration are both necessary for SCI repair. In this study, we evaluated the co-transplantation of olfactory ensheathing cells (OEC) and embryonic stem (ES) cell-derived motor neurons (ESMN) on contused SCI. Methods: OEC...
متن کاملZebrafish motor neuron subtypes differ electrically prior to axonal outgrowth.
Different muscle targets and transcription factor expression patterns reveal the presence of motor neuron subtypes. However, it is not known whether these subtypes also differ with respect to electrical membrane properties. To address this question, we studied primary motor neurons (PMNs) in the spinal cord of zebrafish embryos. PMN genesis occurs during gastrulation and gives rise to a heterog...
متن کاملrepair of spinal cord injury by co-transplantation of embryonic stem cell-derived motor neuron and olfactory ensheathing cell
background: the failure of regeneration after spinal cord injury (sci) has been attributed to axonal demyelination and neuronal death. cellular replacement and white matter regeneration are both necessary for sci repair. in this study, we evaluated the co-transplantation of olfactory ensheathing cells (oec) and embryonic stem (es) cell-derived motor neurons (esmn) on contused sci. methods: oec ...
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Hypertonia and hyperreflexia are classically described responses to upper motor neuron injury. However, acute hypotonia and areflexia with motor deficit are hallmark findings after many central nervous system insults such as acute stroke and spinal shock. Historic theories to explain these contradictory findings have implicated a number of potential mechanisms mostly relying on the loss of desc...
متن کاملEndogenous Mechanisms of Neuroprotection and Neuroregeneration
481 Fom the point of view of oligodendrocyte and demyelinated axon interaction, glial scars of reactive astrocytes obstruct to myelinating oligodendrocytes the access to naked axons. On the other hand, astrocytes also produce neurotrophic factors that promote remyelination. An alternative strategy that CNS cells have to avoid axonal damage, when remyelination is not efficient, consists in that ...
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ژورنال
عنوان ژورنال: Journal of Neuroimmunology
سال: 2007
ISSN: 0165-5728
DOI: 10.1016/j.jneuroim.2006.11.029